Buy The Strokes tickets from the official Ticketmaster.com site. Find The Strokes tour schedule, concert details, reviews and photos.Mind Your Risks. Alzheimer’s disease is the most common form of dementia. There are drugs that may improve the quality of life for people who have it, but there is no cure for the disease. Vascular dementia is caused by “silent strokes,” also called infarcts (tissue damage), that can often go unnoticed. Damage to the brain, as a result of multiple strokes over time, gradually leads to a loss of brain function. Moreover, population- based autopsy studies have shown that many patients who die with dementia often have a combination of both Alzheimer’s pathology and brain injury due to vascular disease or silent strokes, often called “mixed. Experts now believe that the processes that give rise to vascular disease in the brain and Alzheimer’s disease may converge, dramatically increasing the likelihood and severity of dementia more than either condition. Strokes can cause a host of cognitive disabilities, including effects on memory, speech and language, and everyday problem solving. But even without suffering an obvious stroke, individuals at risk for stroke may experience cognitive impairment. Silent strokes and vascular damage to the “wires” that connect brain regions cause diffuse white matter disease. These brain changes. Learn more about dementia at our NINDS Dementia Information Page. There, you can also find out about other organizations that provide. Learn more about some of the research on high blood pressure, dementia, and stroke. ![]() NINDS Know Stroke Campaign - strokechallenges. Contents. Introduction. Stroke Basics. Ischemic Stroke and Transient Ischemic Attack. Hemorrhagic Stroke. Vascular Cognitive Impairment. ![]() Stroke Diagnostics and Brain Imaging. Stroke Risk Factors. Unmodifiable Risk Factors. Modifiable Risk Factors. Current Stroke Treatment and Prevention. Medications. Surgeries and Other Procedures. Rehabilitation. Research and Hope for the Future. Prevention. Acute Stroke and Post- Stroke Therapy. Neuroprotection. Changing Stroke's Impact by Changing Attitudes. Introduction. Chances arethat nearly everyone... Research sponsored by the National Institutes of Health (NIH) shows that about one in six Americans will experience a stroke at some point after age 6. Stroke is fatal in about 1.
Listen to songs from the album Future Present Past EP, including 'Drag Queen,' 'Oblivius,' 'Threat of Joy,' and many more. Buy the album for $3.99. Songs start at $1.29. Free with Apple Music subscription. ![]() ![]() ![]() These trends have made stroke the third leading cause of death in the U. S. (behind heart disease and cancer) and a major cause of disability. A stroke is a sudden event affecting the brain's blood supply. In an ischemic stroke, a blood vessel that supplies the brain becomes blocked. In a hemorrhagic stroke, a blood vessel in the brain bursts. Although stroke is most common in older people, it can occur in individuals of any age, including young adults, children, infants, and fetuses in the womb. Fortunately, the prospects for preventing and treating stroke are far better than they were just a few decades ago, thanks in part to research supported by the NIH's National Institute of Neurological Disorders and Stroke (NINDS). NINDS- funded investigators have identified many risk factors for stroke, such as hypertension (high blood pressure), arming people with the ability to reduce their risk through lifestyle changes or medication. Clinical trials supported by NINDS also led to the only drug approved by the Food and Drug Administration specifically for treating acute ischemic stroke- the "clot- buster" t. PA. When given within three hours of stroke onset, the drug significantly reduces the likelihood of long- term disability. Current NINDS priorities in stroke research are set by its Stroke Progress Review Group (SPRG), an advisory body made up of scientists, clinicians, patient advocates, and industry representatives. One such priority is to develop new and more effective treatments for acute stroke. NINDS is supporting research on novel ways to restore blood flow to the ischemic brain, as well as treatments that would act directly on brain cells to protect them from the effects of stroke. Another priority is to improve post- stroke recovery by tapping into the brain's capacity to repair itself; for example, by using drugs to stimulate the growth of new brain cells or physical therapy regimens to stimulate the re- wiring of existing cells. Finally, stroke prevention and the identification of stroke risk factors remain important parts of the NINDS portfolio. NINDS supports several complementary networks of research centers aimed at improving treatment and care for individuals with acute stroke. The Specialized Program of Translational Research in Acute Stroke (SPOTRIAS) helps move experimental therapies for acute stroke from the lab into early- phase clinical studies. SPOTRIAS sites are located at major academic medical centers across the country, where scientists studying the molecular and cellular mechanisms of stroke can work closely with clinicians who have expertise in acute stroke care. The Neurological Emergencies Treatment Trials (NETT) network creates a similar collaborative framework among neurologists, neurosurgeons, and emergency medicine physicians to facilitate clinical trials on stroke in the emergency room. Meanwhile, through the Stroke Preclinical Trials Consortia, NINDS is forging collaboration among investigators who are testing potential stroke treatments in animal models. Finally, NINDS outreach programs help bridge research and practice by educating individuals, their loved ones, and healthcare providers about the latest advances in treatment and prevention. An ischemic stroke occurs when a blood vessel supplying the brain becomes blocked, as by a clot. A hemorrhagic stroke occurs when a blood vessel bursts, leaking blood into the brain. NINDS is supportingresearch on novel ways to restore blood flow to the ischemic brain, as well as treatments that would act directly on brain cells to protect them from the effects of stroke. Stroke Basics. The effects of stroke manifest themselves rapidly. The five most common symptoms of stroke are: Sudden weakness or numbness of the face or limbs, especially on one side of the body. Sudden confusion or difficulty speaking or understanding speech. Sudden trouble seeing in one or both eyes. Sudden trouble walking, dizziness, or loss of balance or coordination. Sudden severe headache with no known cause. The exact symptoms depend on where in the brain's vascular system the blockage or rupture has occurred. Strokes that predominantly affect one hemisphere of the brain are common. Since each hemisphere controls the opposite side of the body, a stroke in the left hemisphere will cause motor and sensory deficits on the right side of the body, and vice versa. The long- term outcomes after a stroke vary considerably and depend partly on the type of stroke and the age of the affected person. Although most stroke survivors regain their functional independence, 1. Some will experience a permanent decline in cognitive function known as post- stroke or vascular dementia. Unfortunately, many stroke survivors face a danger of recurrent stroke in the future. About 2. 0 percent of people who experience a first- ever stroke between ages 4. Finding treatments to help prevent stroke in this high- risk group is a major focus of NINDS- supported research. Ischemic Stroke and Transient Ischemic Attack. Ischemic strokes make up about 8. Just as a heart attack occurs when there is insufficient blood flow to the heart, an ischemic stroke (sometimes called a "brain attack") occurs when there is a sudden interruption in blood flow to one or more regions of the brain. Like all cells in the body, neurons and other brain cells require oxygen and glucose delivered through the blood in order to function and survive. A few minutes of oxygen deprivation- called ischemia- is enough to kill millions of neurons. Moreover, ischemia can provoke inflammation, swelling (edema), and other processes that can continue to cause damage for hours to days after the initial insult. Obstructive blood clots are the most common cause of ischemic stroke. Clotting (or coagulation) is a vital function that helps prevent bleeding when a blood vessel is damaged, but clots can also obstruct normal blood flow. When a clot forms in association with the wall of a blood vessel and grows large enough to impair blood flow, it is called a thrombus; a clot that breaks off the vessel wall and travels through the blood is an embolus. A cardioembolic stroke is caused by a clot that originates in the heart. Cardiac emboli are most likely to form in people with heart conditions such as atrial fibrillation (AF, an irregular heartbeat), heart failure, stenosis, or infections within the valves of the heart. They may also occur post- heart attack. Another contributor to ischemic stroke is chronic atherosclerosis, which is a buildup of fatty deposits and cellular debris (plaque) on the inside of the blood vessel wall. As atherosclerotic plaques grow, they cause narrowing of the blood vessel (a condition called stenosis). Atherosclerosis can also activate cells involved in clotting. Medical Terms for Common Stroke Symptoms and Disabilities. Hemiplegia: paralysis on one side of the body. Hemiparesis: weakness on one side of the body. Hemineglect: lack of awareness of one side of the body or one side of the visual field. Dysphagia: difficulty swallowing. Dysarthria: difficulty talking, caused by facial weakness. Aphasia: difficulty speaking or understanding speech, caused by damage to the brain's language centers. Immediately after an ischemic stroke, the brain usually contains an irreversibly damaged core of tissue and an area of viable but at- risk tissue called the ischemic penumbra. Restoring normal blood flow- a process known as reperfusion- is essential to rescuing the penumbra. The longer reperfusion is delayed, the more cells in the penumbra will die. The region of brain tissue that is finally damaged is called an infarct. If a stroke were a storm, a transient ischemic attack (TIA), or "mini- stroke," would be an ominous thunderclap. Symptoms of a TIA are similar to those of a full- blown stroke but resolve within 2. Still, the short- lived nature of TIAs does not mean that they leave the brain unharmed. In about 4. 0 to 5. TIA, a tiny dot of infarct can be seen by brain imaging. Even when there is no sign of brain infarction, a TIA is both a warning and an opportunity for intervention. While someone who has experienced a full- blown stroke has a two to seven percent risk of having another stroke within the next 9. TIA is 1. 0 to 2. In many cases, TIAs may be caused by an unstable clot that could create a more permanent blockage within the brain's blood supply at any moment. Fortunately, there are a variety of treatments that can reduce the risk of stroke following a TIA, including medications to lower blood pressure and inhibit blood clotting. If necessary, surgical procedures can clear away plaque in the arteries that supply the brain, or a procedure called stenting can be used to widen the arteries. Severe strokes could be avoided if more people sought medical attention after a TIA. For more about these treatments, see the chart on p. Current Stroke Treatment and Prevention" on p. Hemorrhagic Stroke. An intracerebral hemorrhage occurs when a blood vessel ruptures within the brain. Several conditions can render blood vessels in the brain prone to rupture and bleeding. Chronic hypertension and a condition known as cerebral amyloid angiopathy can weaken the blood vessel wall.
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